By Ramana S. Moorthy, MD
A dialogue of the scientific method of uveitis results in largely rewritten chapters on noninfectious (autoimmune) and infectious varieties of uveitis. additionally lined are endophthalmitis, masquerade syndromes, and problems of uveitis. A dialogue on ocular involvement in AIDS has been up-to-date. The part on immunology describes the human immune reaction in phrases that make it hugely obtainable to readers.
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Extra info for 2008-2009 Basic and Clinical Science Course: Section 9: Intraocular Inflammation and Uveitis (Basic and Clinical Science Course 2008-2009)
CHAPTER 2: Immunization and Adaptive Immunity. 19 B lymphocytes, and T lymphocytes) into efferent lymphatics and venous circulation. The intent of the immune system is conveyed back to the original site, where an effector response occurs (ie, immune complex formation or delayed hypersensitivity reaction). The following discussion covers the important aspects of each phase in more detail. Phases of the Immune Response Arc Afferent Phase The initial recognition, transport, and presentation of antigenic substances to the adaptive immune system constitute the afferent phase of the immune response arc.
The anterior chamber is a fluid-filled cavity; circulating aqueous humor provides a unique medium for intercellular communication among cytokines, immune cells, and resident tissue cells of the iris, ciliary body, and corneal endothelium. 0% of total serum protein concentration), even normal aqueous humor contains a complex mixture of biological factors, such as immunomodulatory cytokines, neuropeptides, and complement inhibitors, that can influence immunologic events within the eye. CHAPTER 3: Ocular Immune Responses.
Martin S. Burton D. Roitt 1M. Roitt's Essential Immunology. 11th ed. Malden. MA: Blackwell; 2006. Male OK, Cooke A. Owen M, Trowsdale J, Champion B. Advanced Immunology. 3rd ed. St Louis: Mosby; 1996. CLINICAL EXAMPLES 2-1 Primary response to poison ivy toxin The first contact between the poison ivy resin urushiol and the epidermal surface of an exposed extremity, such as the forearm, triggers the immunologic mechanisms of poison ivy dermatitis. The afferent phase of this primary response begins when the toxin permeates into the epidermis, where much of it binds to extracellular proteins, forming a protein-toxin conjugate technically called a hapten.
2008-2009 Basic and Clinical Science Course: Section 9: Intraocular Inflammation and Uveitis (Basic and Clinical Science Course 2008-2009) by Ramana S. Moorthy, MD